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LPS- and LTA-Induced Expression of IL-6 and TNF-α in Neonatal and Adult  Blood: Role of MAPKs and NF-κB
LPS- and LTA-Induced Expression of IL-6 and TNF-α in Neonatal and Adult Blood: Role of MAPKs and NF-κB

IL-10 inhibits TNF-α production in LPS-stimulated RAW 264.7 cells. (A)... |  Download Scientific Diagram
IL-10 inhibits TNF-α production in LPS-stimulated RAW 264.7 cells. (A)... | Download Scientific Diagram

Atorvastatin increases lipopolysaccharide‑induced expression of tumour  necrosis factor‑α‑induced protein 8‑like 2 in RAW264.7 cells
Atorvastatin increases lipopolysaccharide‑induced expression of tumour necrosis factor‑α‑induced protein 8‑like 2 in RAW264.7 cells

PLOS ONE: Naringin Decreases TNF-α and HMGB1 Release from LPS-Stimulated  Macrophages and Improves Survival in a CLP-Induced Sepsis Mice
PLOS ONE: Naringin Decreases TNF-α and HMGB1 Release from LPS-Stimulated Macrophages and Improves Survival in a CLP-Induced Sepsis Mice

Inhibition of Prolyl Hydroxylase Domain-Containing Protein Suppressed  Lipopolysaccharide-Induced TNF-α Expression | Arteriosclerosis, Thrombosis,  and Vascular Biology
Inhibition of Prolyl Hydroxylase Domain-Containing Protein Suppressed Lipopolysaccharide-Induced TNF-α Expression | Arteriosclerosis, Thrombosis, and Vascular Biology

Augmented Lipopolysaccharide-Induced TNF-α Production by Peritoneal  Macrophages in Type 2 Diabetic Mice Is Dependent on Elevated Glucose and  Requires p38 MAPK | The Journal of Immunology
Augmented Lipopolysaccharide-Induced TNF-α Production by Peritoneal Macrophages in Type 2 Diabetic Mice Is Dependent on Elevated Glucose and Requires p38 MAPK | The Journal of Immunology

PLOS ONE: A Novel Strategy for TNF-Alpha Production by 2-APB Induced  Downregulated SOCE and Upregulated HSP70 in O. tsutsugamushi-Infected Human  Macrophages
PLOS ONE: A Novel Strategy for TNF-Alpha Production by 2-APB Induced Downregulated SOCE and Upregulated HSP70 in O. tsutsugamushi-Infected Human Macrophages

Whole-body deletion of LPS-induced TNF-α factor (LITAF) markedly improves  experimental endotoxic shock and inflammatory arthritis | PNAS
Whole-body deletion of LPS-induced TNF-α factor (LITAF) markedly improves experimental endotoxic shock and inflammatory arthritis | PNAS

Progranulin inhibits LPS-induced macrophage M1 polarization via NF-кB and  MAPK pathways | BMC Immunology | Full Text
Progranulin inhibits LPS-induced macrophage M1 polarization via NF-кB and MAPK pathways | BMC Immunology | Full Text

Molecules | Free Full-Text | 5-Hydroxymethylfurfural Mitigates  Lipopolysaccharide-Stimulated Inflammation via Suppression of MAPK, NF-κB  and mTOR Activation in RAW 264.7 Cells | HTML
Molecules | Free Full-Text | 5-Hydroxymethylfurfural Mitigates Lipopolysaccharide-Stimulated Inflammation via Suppression of MAPK, NF-κB and mTOR Activation in RAW 264.7 Cells | HTML

Inhibition of transmembrane TNF-α shedding by a specific antibody protects  against septic shock | Cell Death & Disease
Inhibition of transmembrane TNF-α shedding by a specific antibody protects against septic shock | Cell Death & Disease

LPS-induced TNF-α factor (LITAF)-deficient mice express reduced LPS-induced  cytokine: Evidence for LITAF-dependent LPS signaling pathways | PNAS
LPS-induced TNF-α factor (LITAF)-deficient mice express reduced LPS-induced cytokine: Evidence for LITAF-dependent LPS signaling pathways | PNAS

IJMS | Free Full-Text | Cordycepin Inhibits Lipopolysaccharide (LPS)-Induced  Tumor Necrosis Factor (TNF)-α Production via Activating AMP-Activated  Protein Kinase (AMPK) Signaling
IJMS | Free Full-Text | Cordycepin Inhibits Lipopolysaccharide (LPS)-Induced Tumor Necrosis Factor (TNF)-α Production via Activating AMP-Activated Protein Kinase (AMPK) Signaling

Lipopolysaccharide-induced tumor necrosis factor-α factor enhances  inflammation and is associated with cancer (Review)
Lipopolysaccharide-induced tumor necrosis factor-α factor enhances inflammation and is associated with cancer (Review)

Combinations of ERK and p38 MAPK Inhibitors Ablate Tumor Necrosis Factor-α ( TNF-α) mRNA Induction - Journal of Biological Chemistry
Combinations of ERK and p38 MAPK Inhibitors Ablate Tumor Necrosis Factor-α ( TNF-α) mRNA Induction - Journal of Biological Chemistry

Lipopolysaccharide-induced tumor necrosis factor-α factor enhances  inflammation and is associated with cancer (Review)
Lipopolysaccharide-induced tumor necrosis factor-α factor enhances inflammation and is associated with cancer (Review)

Lipopolysaccharide-Induced Tumor Necrosis Factor Alpha Production by Human  Monocytes Involves the Raf-1/MEK1-MEK2/ERK1-ERK2 Pathway | Infection and  Immunity
Lipopolysaccharide-Induced Tumor Necrosis Factor Alpha Production by Human Monocytes Involves the Raf-1/MEK1-MEK2/ERK1-ERK2 Pathway | Infection and Immunity

Inhibition of LPS-Induced TNF-{Alpha} and NO Production in Mouse Macrophage  and Inflammatory Response in Rat Animal Models by a Novel Ayurvedic  Formulation, BV-9238 - Global Medical Discovery
Inhibition of LPS-Induced TNF-{Alpha} and NO Production in Mouse Macrophage and Inflammatory Response in Rat Animal Models by a Novel Ayurvedic Formulation, BV-9238 - Global Medical Discovery

LPS induces the interaction of a transcription factor, LPS-induced TNF-α  factor, and STAT6(B) with effects on multiple cytokines | PNAS
LPS induces the interaction of a transcription factor, LPS-induced TNF-α factor, and STAT6(B) with effects on multiple cytokines | PNAS

TNF-α Induction by LPS Is Regulated Posttranscriptionally via a  Tpl2/ERK-Dependent Pathway - ScienceDirect
TNF-α Induction by LPS Is Regulated Posttranscriptionally via a Tpl2/ERK-Dependent Pathway - ScienceDirect

Lipopolysaccharide-Induced Tumor Necrosis Factor Alpha Production by Human  Monocytes Involves the Raf-1/MEK1-MEK2/ERK1-ERK2 Pathway | Infection and  Immunity
Lipopolysaccharide-Induced Tumor Necrosis Factor Alpha Production by Human Monocytes Involves the Raf-1/MEK1-MEK2/ERK1-ERK2 Pathway | Infection and Immunity

Lipopolysaccharide and Tumor Necrosis Factor Alpha Inhibit Interferon  Signaling in Hepatocytes by Increasing Ubiquitin-Like Protease 18 (USP18)  Expression | Journal of Virology
Lipopolysaccharide and Tumor Necrosis Factor Alpha Inhibit Interferon Signaling in Hepatocytes by Increasing Ubiquitin-Like Protease 18 (USP18) Expression | Journal of Virology

β-Ionone attenuates LPS-induced pro-inflammatory mediators such as NO, PGE2  and TNF-α in BV2 microglial cells via suppression of the NF-κB and MAPK  pathway - ScienceDirect
β-Ionone attenuates LPS-induced pro-inflammatory mediators such as NO, PGE2 and TNF-α in BV2 microglial cells via suppression of the NF-κB and MAPK pathway - ScienceDirect